Our immune system is an important part of the body, as it is responsible for protecting our body from various attacks harmful viruses, bacteria and other microorganisms. The immune system consists of a complex network of white blood cells, antibodies and immune factors to heal the body when we are pain and protects the body against cancer and infections. It acts as a fence of the house and keeps intruders like germs and contaminants out. However, there are times when the line of defense goes awry and begins attacking the body itself. This is how you soldiers fighting for their country and for some reason they are wobbly and starting to shoot at his men of the country. It is a condition where the immune system correctly understands the body to be his enemy and begins to attack is called a hyperactive immune system. An overactive immune system is nothing but dysfunction of the immune system, also called autoimmune or autoimmune diseases. An overactive immune system leads to autoimmune diseases and allergies due to imbalance in white blood cells of the body and other immune factors. This imbalance causes hyperactivity of the immune components and, when these components are not able to distinguish between enemies and its own cells. An overactive immune system over time results in various autoimmune diseases like multiple sclerosis, in which the immune system attacks the myelin sheath, and arthritis when the immune system attacks the synovial membrane located between the joints. Other autoimmune disease lupus, psoriasis, fibromyalgia, inflammatory bowel disease, etc. However, why the immune system goes awry, is still unknown. Although genetics is one of the reasons we do not have caused the disease was still cause an overactive immune system in the family. When the body attacks its own cells and tissues, they lead to localized inflammation in the area where the attack and can cause various symptoms. For example, if the immune system in a state of dysfunctional attacking the synovial membrane present in the joints, inflammation, it will happen to these compounds, along with the pain. As the attack continues it will lead to arthritis and one will experience early symptoms of arthritis. It mainly consists of the symptoms of pain and inflammation in the area attacked.
Until recently the most common overactive immune system treatment was administered steroids, chemotherapy and major immunosuppressants. However, these drugs are held in various side effects and risk to the body. Last, effective treatments are aimed only at those parts of the immune system that do not function. They do this by blocking specific molecules in inflammatory way. Autologous hematopoietic stem cell transplantation (HSCTs), known to be the most promising measures to achieve long-term remission of autoimmune diseases. These transplant involves removing the dysfunctional immune system cells and replace them with fresh cells. Autoimmune diseases affect approximately 5% to 8% of the population, of which about 75% of autoimmune diseases falls on women and the fourth leading cause of disability in women. In addition, researchers from the University of Leicester say that hyperactive immune system can cause cancer. In autoimmune diseases have lasix drug reactions no treatment, as now, these women end up carrying the burden of this difficult disease for life. The only thing they can do is eat an overactive immune system diet and take medication such as anti-inflammatory drugs and steroids to relieve the pain. .Friday, February 24, 2012
This effect is well known in premenopausal ...
Get the disease. The prevalence of osteoporosis in men not as often as women, but depending on age and other risk factors, it is not rare. There are many people living with osteoporosis. Osteoporosis is a disease of bone that leads to bone loss. This loss of bone mass leads to
bones become porous and brittle. People with osteoporosis are at risk severe fractures due to this fragile state of the bones. In the elderly,
, these cracks can be life threatening. So, what causes osteoporosis in men? Well, there are two major factors that contribute to bone loss in men. First of all, people are faced with >> << lower bone mass with age (as well as women, but usually not so great). Second, people with average risk factors for osteoporosis may be additional losses in the
, which can lead to osteoporosis. Aging causes of osteoporosis in men, too! Men suffer from reduced bone mass with age, but the effect is not as dramatic as in women. After menopause, women are at much greater risk for
osteoporosis by reducing estrogen levels (estrogen has a protective effect on bone health). Men also requires a certain level of sex hormones for
healthy bones, but they do not face a reduction in these hormones later in life - for men, sex hormones decrease after 65 years (women may suffer >> << hormone reduction of 40!). Moreover, most men have more bone mass primary than in women. Peak bone mass in men is usually achieved somewhere in the middle
twenties, where it stabilized for several years and begins to decline around the mid thirties. The pace of decline is estimated to range from 0. 5
1. 0% on average per year. So, let's delve into the reasons why aging causes osteoporosis in men. Aging affects the vitamin D in the skin. Vitamin D is crucial for bone health. Why? Without enough vitamin D, calcium we swallows a hard time to be absorbed. And calcium
required for the formation of bone tissue. Our most important source of vitamin D is sun. When our skin is exposed to ultraviolet sun rays, a form of cholesterol (7-dehidroholesteryn)
in our skin becomes a pre-vitamin D, which is then converted to vitamin D. As we age, our skin begins to lose the ability to produce this >> << cholesterol - so that people in the age of 70 can produce only 20% as much as men in the twenties. Since vitamin D is crucial for calcium absorption >> << This effectively reduces the amount of calcium elderly person has to form bone. This age-related decline in vitamin D is, of course, the risk of >> << factor of osteoporosis in men and women. Aging affects the levels of parathyroid in turn affect the amount of calcium in the bones. With age, levels of parathyroid hormone (PTH) in our body increases. Parathyroid hormone has only one role - to regulate calcium levels in the blood
exact reason why PTH increases with age is not well understood. However, when PTH high, a signal for our body to
. increase the calcium in the blood for use in important physiological activity (muscle contraction, cardiac regulation and nerve transmission). Where calcium come from? Unfortunately, it comes from our bones. When calcium is needed for other functions, he pulled from the bones >> << and deposited into the bloodstream. Elevated levels of PTH is a signal to the removal of calcium (known as resorption). As more and more calcium
pull from the bones, they become more brittle. That is why increased parathyroid hormone (which occur with age in both sexes)
promote osteoporosis in men and women. Hormonal changes in men affect health support-Too. Men produce estrogen - is actually converted to testosterone via the enzyme aromatase. However, men are only about one-tenth the level of estrogen >> << compared with pre-menopausal women. With age in men, their estrogen levels decrease, as well as in women. For relative scale, however, decrease
men are much less severe than in women. Studies have shown that even in men, however, estrogen plays an important role in bone >> << density by inhibiting osteoclast activity. Osteoclasts are cells that breakdown bone, which allows calcium to be released into the bloodstream. This bone breakdown occurs in the proper balance >> << with the bone building activity is very important for good general health and physical work. However, if the decrease in estrogen levels causes
activity of osteoclasts (which break down bone) ahead of osteoblasts (which build bone), bone mineral density decreases. This effect is well known in premenopausal, but low levels of estrogen in men (especially older people) lead to bone loss mineral too. men too, as they age. The level of testosterone usually decreases with age in men (especially older than 65 years). Recognized that lower testosterone levels can lead to
osteoporosis in men. Testosterone is thought to promote the activities of osteoblasts and osteoclasts brake. The combination of low levels of sex hormones (estrogen and testosterone) in men with age reduces bone mass. Some studies show that
This is the ratio of these two hormones that affect the incidence of osteoporosis furosemide in men. Men have two distinct advantages over women - their level of hormones
not fall later in life, and they usually have more initial mass of bone than women. Other factors besides the normal aging process. Secondary osteoporosis is osteoporosis, due to factors other than the normal aging process. These reasons are usually the result of certain
prescription drugs, chronic illness and / or lifestyle factors risk. For more information on these risk factors for osteoporosis, please refer to my page. So, below are some of the major risk factors >> << to secondary osteoporosis in men:
glucocorticoid drugs - These steroids have serious consequences for bone health and a major cause of secondary osteoporosis in men and women. Alcoholism - Excessive alcohol consumption affects many important bone-building, especially the liver's ability to convert vitamin D
(where it is stored) in the biologically active form. Lack of physical activity - exercise should be emphasized bones, which leads to an increase in bone formation. Get too little and bone health is suffering. Back to my page >> << to learn more about the benefits of physical activity! Smoking - nicotine products lower levels of sex hormones, especially estrogen, which plays an important role in suppressing the activity of osteoclasts. For more information about
how this habit affects bone health, check out my page. Chronic diseases - Some diseases such as chronic kidney disease,, and hyperparathyroidism (to name just a few), can lead to osteoporosis
, . Hypogonadism - This condition leads to a decrease in testosterone levels, which negatively affects bone mineral density. How is osteoporosis treated in men? Treatment of osteoporosis in men is very different from the treatment of osteoporosis in women. For those who are in danger, but not yet suffering from advanced osteoporosis >> << additives recommended (read my page >> <<). Applications include strontium, calcium and vitamin D (check with your doctor first
relatively calcium - some studies show high levels of calcium may be associated with prostate problems). Healthy exercise and proper nutrition for the prevention of osteoporosis is also extremely important - please refer to my page, read on >> << other natural treatment of osteoporosis. In the case of more severe osteoporosis, or men who have
multiple risk factors for osteoporosis that prescription drugs to treat osteoporosis such as bisphosphonates (Fosomax (R)) may be recommended. Since these prescription drugs osteoporosis can have serious side effects, I hope you bring up yourself and explore other options before you begin
on them. Participation in and after will go a long way toward prevention and treatment
conditions such as. Be sure to check back often - how can I find more information
to help you in creation, I will add it to this site! RN, a certified fitness trainer links
: CG, Soerensen HT-Eriksen EF. The prevalence of low serum estradiol in osteoporosis in men. Osteoporosis International 2000 11 697-701. - Aging is not the only cause of osteoporosis! - Everything You Need to Know About Osteoporosis is a test. - Does smoking effect on bone mineral density? - What you need to know how steroids affect the bone! - What you need to know about the link between these eating disorders and bone loss. - Does drinking coffee put you at risk of osteoporosis? .
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Condition. screening for osteoporosis, in january 2011
Here are the options the U.S. preventive services target (USPSTF) recommendations on screening for osteoporosis. / USPSTF recommends screening for osteoporosis in women aged 65 and older and in young women whose risk of fracture is equal to or greater than 65-year old white woman who does not have any risk factors. Condition. USPSTF concluded that the available data are insufficient to assess the benefit and harms of screening for osteoporosis in men. Condition. Screening for osteoporosis lasix mg iv, in January 2011
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Current as of March 2011 Internet Citation: ....... Screening for osteoporosis, the topic page in January 2011 the U.S. working group on prevention http://www uspreventiveservicestaskforce org / USPSTF / uspsoste HTM
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Stage iii: shortness of breath on mild strain ...
In-Depth from Adam appropriate medications for COPD depend on the stage of severity, as defined symptoms. Global Initiative for chronic obstructive pulmonary disease (GOLD) proposed a strategy that is widely recognized. GOLD categories of COPD severity as follows:
Phase I: smoker's cough, little or no wheezing, no clinical signs of COPD, FEV1 greater than 80% of properly. Stage II: shortness of breath on exertion, cough sputum production, some clinical signs of COPD, FEV1 50 - 80% of the good. Stage III: Shortness of breath on mild pressure, FEV1 30 - 50% of the good. Stage IV: Shortness of breath on mild pressure, right heart failure, cyanosis, FEV1 less than 30% of the good. Gold treatment strategy additives, which means that drugs will be added, not subtracted, and the disease progresses. Treatment includes:
Patients with Gold stage I - IV: Treatment of inhaled anticholinergic and inhaled short-acting beta 2-agonists. Patients with the Golden Phase II - IV: addition of inhaled long-acting bronchodilator in place of short-acting beta 2-agonists. Combining bronchodilators may be used. In patients who do not respond to bronchodilators, slow-release theophylline can be used. Patients with Gold stage III - IV: Regular use of inhaled corticosteroids for those with repeated exacerbations. Systemic corticosteroids for severe exacerbations. Patients with the Golden Stage IV: Long-term oxygen therapy, consideration of surgical techniques. American College of Physicians published revised guidelines for the treatment of COPD, including:
In patients with COPD, reserve treatment for those who have respiratory symptoms and FEV1 less than 60% of the good. Patients with symptoms and FEV1 less than 60% of appropriate, should be treated with long acting inhaled beta-agonists long-acting inhaled anticholinergic or inhaled corticosteroids. A combination of inhalation therapy can be used in these patients. Patients with COPD and the rest of hypoxia should be treated with oxygen therapy. Patients with symptoms and FEV1 less than 50% of the good should consider pulmonary rehabilitation. Quitting smoking. Quitting smoking is the first and most important step in treating COPD and slowing its development. Quitting smoking reduces the symptoms of breathlessness and cough, and risk of fatal and nonfatal heart disease, probably by reducing inflammation. Diets. Good nutrition is always important. Dietary issues become critical in late COPD, when breathing is obstructed. Many patients with COPD lose muscle tone and body weight, and it seems zachahnuty. This may be due to the very effort to breathe rapidly consumes calories. Some patients are difficult to stop the effort of breathing long enough to chew. Dietitian can be extremely useful in finding the right products and design meal plans to help COPD patients to be healthy. There is no convincing evidence in favor of the use of food additives in patients with COPD. This can help avoid sausages, which studies show can increase the risk of developing COPD. Extra oxygen. Supplemental oxygen therapy is an important component of COPD. It can:
All these factors influence, along with easy to exchange carbon dioxide for oxygen. There is some evidence that additional oxygen can also reduce heart problems such as heart rhythm in patients with COPD. Long-term oxygen therapy given continuously through the nose, as shown, continue to exist in as much as 30%. Blending oxygen with nitrogen (helyoks) showed that more effectively than additional oxygen only in improving endurance and exercise capacity. Pulmonary rehabilitation. Pulmonary rehabilitation is a proven method of dismissal difficulty breathing (dyspnea), reduced hospitalizations and disability resulting from COPD and improve mental and lasix heart rate physical quality of life, although there is no evidence that it improves survival. Treatment is recommended for patients with stable chronic lung disease that significantly affect respiratory symptoms. Many hospitals offer these programs led by a team of specialists in health. Pulmonary rehabilitation is designed for individual patients, but usually include:
Programs usually last 6 - 12 weeks, but appears more programs to ensure more sustainable benefits. Program service may slightly improve the long-term results. Exercise. Exercise is very important to maintain strength and endurance, both of which strongly depends on COPD. Weight bearing exercises are important to maintain the quality of life and ability to live independently. For greater benefits, programs must be combined with low and high intensity exercise power and endurance. The use of noninvasive ventilation (NIPPV) during exercise provides small, very short-term benefits. Receiving supplemental oxygen during rehabilitation exercises can improve the endurance of patients. There is no evidence that training muscles inhalation is effective in pulmonary rehabilitation. Surgery. When the patient does not respond to medication, surgery becomes a possible option. Selection includes:
The goal of treatment of COPD, in addition to relief of symptoms is to prevent exacerbations. Each exacerbation causes lung function decline. Bringing lung function to its pre-exacerbation state can take 6 months. When exacerbations often lung function may never return to normal, and the patient's condition spirals downhill. Recent studies have shown that levels of markers of inflammation in the body rise dramatically during exacerbations and in proportion to the severity of exacerbation. This may mean that the aggravation associated with inflammation in the body, not just in the lungs themselves. Measurement of C-reactive protein (CRP), a marker of inflammation, which is used to confirm exacerbations, predict their severity and helps determine the prognosis. Aggravation usually caused by bacterial or viral infections or air pollution. The reason never mentioned in 1/3 of patients. Oxygen. Supplemental oxygen with controlled oxygen therapy and noninvasive ventilation with positive pressure. Bronchi. Inhaled anticholinergics or short-acting beta 2-agonists may be used. Theophylline is not recommended because it provides very little benefit and carries a risk of serious side effects. Corticosteroids. This can be provided by or through a vein (intravenously) or orally (per os) for up to 2 weeks. This treatment is only possible when patients did not receive long-term oral corticosteroids. Antibiotics. They can be used if signs of infection such as fever or yellow or green phlegm. .
Primary osteoporosis can be of two main ...
Abnormal loss of bone resulting in fragile porous bones due to lack of the most common in postmenopausal women, so that is extremely porous. Reducing weight without changes in the composition, leading to fractures. Primary osteoporosis can be of two types: postmenopausal osteoporosis (osteoporosis in postmenopausal women) and age related or senile osteoporosis. Everyone who works in the medical industry is sometimes necessary to know how to define the word in medical terminology. For example - how to explain osteoporosis? Here you can see the medical definition of osteoporosis. Medical Dictionary. cc your online buy lasix 40 mg dictionary, full medical definition. .
In fact >> <<, a man who in
bullous emphysema are very similar to ordinary emphysema. All of them are related to the body
unable to take in as much oxygen as expected. One of the main >> << symptoms shortness of breath. A person may find that they are not
walk up stairs without getting winded. Shortness of breath, cough and sputum over >> << all symptoms of bullous emphysema. A person may also find that
they no longer have the appetite and start losing weight unintentionally. Feeling of weakness and fatigue is also characteristic when it comes to bullous emphysema. Once a person is diagnosed lasix 500 mg with bullous emphysema, they are put on treatment
plan, which may consist of proper diet and light exercise if possible
. The aim of any treatment of bullous emphysema, to try to enlarge
air to the lungs. Some people with advanced bullous emphysema >> << can not exercise at all. A person can be directed to the
to take steroids. If they are unable to translate the spirit, because their condition
, they can put on the oxygen cylinder, which is a machine that essentially
breathes on them. If a person can not eat through
bullous emphysema them, they can eat intravenously. Last resort for treatment
bullous emphysema is lung transplant. If the light strongly damaged
sufficient reason for removal, and lung transplant will not. And death, and infection may be possible that doctors do not like to do
it, no other treatment options bullous emphysema. Bullous emphysema as well
deadly as regular emphysema. It should be borne in mind that smoking is the main reason >> << for bullous emphysema. A person who smokes will be charged
leave immediately after their diagnosis of bullous emphysema. Best
treatment plan may not help at all if people are still smoking. If a person >> << quit immediately, they tend to have higher life expectancy than people
, who continue to smoke. Although bullous emphysema not curable, individual
early she could live comfortably. In fact >> <<, a man who in an early stage bullous emphysema who is
smoking can have the same life as any other man. Bullous emphysema can be fatal and individuals, in whom it should take a good >> << take care of themselves. Proper treatment can help people to live comfortably, but
early diagnosis is very important. .
Anabolism of protein needed for muscle ...
Anabolism of protein necessary for muscle strength, growth and recovery. For bodybuilders anabolism process of recovery had been called protein synthesis. Synthesis of protein, which organizes itself with nitrogen in proteins by RNA and other enzymes. This process is essential for muscle growth. To and you want to increase protein synthesis. For maximum protein synthesis can increase the growth of muscle mass and strength. This process also includes training, nutrition and rest.
The best way to increase anabolism through what we eat. The process occurs in connection with the explosion of amino acids because we eat, which is focused on our muscles to rebuild themselves and grow in our recovery period. The explosion is called protein throbbing. Proteins pulsing can come from food or protein supplements. It complements anabolic nutrients. This may include notification drink protein workout. Quick burst of protein or amino acid pulse, which makes anabolic stimulation. When we wake up in the morning our body in a catabolic state. During the catabolic state of our muscles are broken (amino acids) to nourish the other cells. The consumption of protein or protein drink when we get up to reverse the process. Metabolism usually contains anabolism and catabolism. Catabolism in response to stress. Process inhibits the synthesis of protein and amino acids used in the storage body. If the process takes too long it can damage the body and cause extensive tissue collapse. Catabolic breakdown of tissues can occur in any organ system. This can be reversed through diet and supplements as needed. How to Fix a catabolic state? You need rest and sleep a lot. Reducing the drug lasix stress in your life. Exercise reduces stress. Eat a healthy diet. Eat plenty of protein. Additives are sometimes necessary to restore anabolism. It is important to remain in a state of anabolism, as a body builder. It you take a protein drink after a workout your body will use it during the day. The maximum density of the anabolic to growth and recovery. This difference between anabolism and catabolism. .